Bummed out
Seth Abrutyn & Anna Mueller
American Sociological Review, forthcoming
Abstract:
Durkheim argued that strong social relationships protect individuals from suicide. We posit, however, that strong social relationships also have the potential to increase individuals’ vulnerability when they expose people to suicidality. Using three waves of data from the National Longitudinal Study of Adolescent Health, we evaluate whether new suicidal thoughts and attempts are in part responses to exposure to role models’ suicide attempts, specifically friends and family. We find that role models’ suicide attempts do in fact trigger new suicidal thoughts, and in some cases attempts, even after significant controls are introduced. Moreover, we find these effects fade with time, girls are more vulnerable to them than boys, and the relationship to the role model — for teenagers at least — matters. Friends appear to be more salient role models for both boys and girls. Our findings suggest that exposure to suicidal behaviors in significant others may teach individuals new ways to deal with emotional distress, namely by becoming suicidal. This reinforces the idea that the structure — and content — of social networks conditions their role in preventing suicidality. Social ties can be conduits of not just social support, but also antisocial behaviors, like suicidality.
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Perceived Control Reduces Mortality Risk at Low, Not High, Education Levels
Nicholas Turiano et al.
Health Psychology, forthcoming
Objective: Both higher levels of educational attainment and a strong sense of control over one’s life independently predict better health and longevity. Evidence also suggests that these 2 factors may combine in multiplicative ways to influence subjective reports of health.
Method: In the Midlife in the United States (MIDUS) national sample (N = 6,135; age = 25 to 75 years), we tested whether stronger beliefs of control over one’s life would moderate the effect of education on 14-year mortality risk.
Results: Proportional hazards modeling indicated that both current levels of education and control beliefs were associated with lower risk of dying, over and above childhood socioeconomic level. In addition, there was a significant interaction between education and control beliefs. Among those low in education, higher control beliefs were associated with a decreased mortality risk. However, at greater levels of education, control beliefs were not associated with mortality risk. This effect remained after adjusting for potential confounding variables, including health behaviors, depressed affect, and general health (chronic illnesses, functional limitations, and self-rated health).
Conclusions: These findings demonstrate the importance of individual perceptions of control in buffering the mortality risk associated with educational disadvantage.
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Tim Kasser et al.
Motivation and Emotion, February 2014, Pages 1-22
Abstract:
Few studies have examined how changes in materialism relate to changes in well-being; fewer have experimentally manipulated materialism to change well-being. Studies 1, 2, and 3 examined how changes in materialistic aspirations related to changes in well-being, using varying time frames (12 years, 2 years, and 6 months), samples (US young adults and Icelandic adults), and measures of materialism and well-being. Across all three studies, results supported the hypothesis that people’s well-being improves as they place relatively less importance on materialistic goals and values, whereas orienting toward materialistic goals relatively more is associated with decreases in well-being over time. Study 2 additionally demonstrated that this association was mediated by changes in psychological need satisfaction. A fourth, experimental study showed that highly materialistic US adolescents who received an intervention that decreased materialism also experienced increases in self-esteem over the next several months, relative to a control group. Thus, well-being changes as people change their relative focus on materialistic goals.
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The Onset of Depression During the Great Recession: Foreclosure and Older Adult Mental Health
Kathleen Cagney et al.
American Journal of Public Health, March 2014, Pages 498-505
Objectives: We examined neighborhood-level foreclosure rates and their association with onset of depressive symptoms in older adults.
Methods: We linked data from the National Social Life, Health, and Aging Project (2005–2006 and 2010–2011 waves), a longitudinal, nationally representative survey, to data on zip code–level foreclosure rates, and predicted the onset of depressive symptoms using logit-linked regression.
Results: Multiple stages of the foreclosure process predicted the onset of depressive symptoms, with adjustment for demographic characteristics and changes in household assets, neighborhood poverty, and visible neighborhood disorder. A large increase in the number of notices of default (odds ratio [OR] = 1.75; 95% confidence interval [CI] = 1.14, 2.67) and properties returning to ownership by the bank (OR = 1.62; 95% CI = 1.06, 2.47) were associated with depressive symptoms. A large increase in properties going to auction was suggestive of such an association (OR = 1.45; 95% CI = 0.96, 2.19). Age, fewer years of education, and functional limitations also were predictive.
Conclusions: Increases in neighborhood-level foreclosure represent an important risk factor for depression in older adults. These results accord with previous studies suggesting that the effects of economic crises are typically first experienced through deficits in emotional well-being.
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George Slavich & Michael Irwin
Psychological Bulletin, forthcoming
Abstract:
Major life stressors, especially those involving interpersonal stress and social rejection, are among the strongest proximal risk factors for depression. In this review, we propose a biologically plausible, multilevel theory that describes neural, physiologic, molecular, and genomic mechanisms that link experiences of social-environmental stress with internal biological processes that drive depression pathogenesis. Central to this social signal transduction theory of depression is the hypothesis that experiences of social threat and adversity up-regulate components of the immune system involved in inflammation. The key mediators of this response, called proinflammatory cytokines, can in turn elicit profound changes in behavior, which include the initiation of depressive symptoms such as sad mood, anhedonia, fatigue, psychomotor retardation, and social-behavioral withdrawal. This highly conserved biological response to adversity is critical for survival during times of actual physical threat or injury. However, this response can also be activated by modern-day social, symbolic, or imagined threats, leading to an increasingly proinflammatory phenotype that may be a key phenomenon driving depression pathogenesis and recurrence, as well as the overlap of depression with several somatic conditions including asthma, rheumatoid arthritis, chronic pain, metabolic syndrome, cardiovascular disease, obesity, and neurodegeneration. Insights from this theory may thus shed light on several important questions including how depression develops, why it frequently recurs, why it is strongly predicted by early life stress, and why it often co-occurs with symptoms of anxiety and with certain physical disease conditions. This work may also suggest new opportunities for preventing and treating depression by targeting inflammation.
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Mismatch or Cumulative Stress: The Pathway to Depression Is Conditional on Attention Style
Esther Nederhof, Johan Ormel & Albertine Oldehinkel
Psychological Science, forthcoming
Abstract:
In the study reported here, the main question we investigated was whether attention style could be a conditional adaptation. We organized participants of the TRacking Adolescents’ Individual Lives Survey (TRAILS; N = 2,230) into shifters, sustainers, and two comparison groups, depending on their performance on a shifting- and a sustained-attention task at age 11 years. Compared with sustainers, shifters reported more pre- and perinatal risk factors and more childhood stress, and they adopted a faster life-history strategy. These differences were not found between the comparison groups, who performed well or poorly on both tasks, which suggests that specialization for either sustained or shifting attention is the key to conditional adaptation. In a subsample (n = 860), we found that stress did not increase depression risk in shifters, whereas a mismatch between early and recent stress predicted depression in sustainers. Cumulative stress predicted depression in the comparison group. These results suggest that shifters retain high levels of plasticity throughout life, whereas sustainers’ adapted their phenotype early in life to the expected mature environment.
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The 5-HTTLPR polymorphism, early and recent life stress, and cognitive endophenotypes of depression
Anne-Wil Kruijt, Peter Putman & Willem Van der Does
Cognition & Emotion, forthcoming
Abstract:
Studies associating interactions of 5-HTTLPR and life adversities with depression have yielded equivocal results. Studying endophenotypes may constitute a more powerful approach. In the current study, it was assessed whether interactions of 5-HTTLPR with childhood emotional abuse (CEA) and recent negative life events (RNLE) affect possible cognitive endophenotypes of depression, namely, attention-allocation bias and the ability to recognise others' mind states in 215 young adults of North-West European descent. The ability to classify others' negative mind states was found to be increased with increasing RNLE in carriers of low-expressing Serotonin Transporter Linked Polymorphic Region (5-HTTLPR) alleles. Carriers of two low-expressing alleles also preferentially oriented attention towards negative information. Gene-environment interactions were not observed for attention allocation bias. No effects involving CEA were observed. These results suggest that low-expressing 5-HTTLPR alleles may confer increased risk for depression through enhanced recognition of negative facial expressions following RNLE.
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Cholesterol and the “Cycle of Violence” in Attempted Suicide
Peter Asellus et al.
Psychiatry Research, forthcoming
Abstract:
An association between low levels of serum cholesterol and violent or suicidal behaviour has frequently been reported. However the role of serum cholesterol in the cycle of violence (Widom, 1989) has not been studied. The aim of this study was to investigate association between exposure to violence during childhood and used adult violence in suicide attempters with low and high serum cholesterol levels. 81 suicide attempters were assessed with the Karolinska Interpersonal Violence Scale (KIVS) measuring exposure to violence and expressed violent behaviour in childhood (between 6–14 years of age) and during adult life (15 years or older). We used median split to dichotomise groups below and above median serum cholesterol. In patients with serum cholesterol below median, the correlation between exposure to violence as a child and used adult violence was significant (rho=0.52, p=0.002), while in patients with serum cholesterol above median, the correlation between exposure to violence as a child and expressed violent behaviour as an adult was not significant (rho=0.25, p=0.2). Comorbid substance abuse predicted violent behaviour as an adult only in patients with serum cholesterol above median. Serum cholesterol may modify the effect of the “cycle of violence”.
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The Effects of Media Violence on Anxiety in Late Adolescence
Anjana Madan, Sylvie Mrug & Rex Wright
Journal of Youth and Adolescence, January 2014, Pages 116-126
Abstract:
Exposure to media violence is related to anxiety in youth, but the causality of the effect has not been established. This experimental study examined the effects of media violence on anxiety, blood pressure, and heart rate in late adolescents. We also examined whether these responses varied by previous exposure to media and real-life violence. College students (N = 209; M age = 18.74; 75 % female; 50 % Caucasian, 34 % African American, 9 % Asian, 3 % Hispanic, and 3 % other racial minorities) were randomized to view either violent or nonviolent high-action movie clips. Participants reported on their anxiety before and after watching the clips, as well as their previous exposure to violence. Measures of blood pressure and heart rate were taken at baseline and during movie viewing. Participants watching violent movie clips showed a greater anxiety increase than those watching nonviolent clips. Both groups experienced increased blood pressure and reduced heart rate during movie watching compared to baseline. Prior exposure to media violence was associated with diminished heart rate response. Additionally, students previously exposed to high levels of real-life violence showed lower blood pressure increases when watching violent clips compared to nonviolent clips. Thus, relatively brief exposure to violent movie clips increased anxiety among late adolescents. Prior exposure to media and real-life violence were associated with lower physiological reactivity to high-action and violent movies, respectively, possibly indicating desensitization. Future studies should investigate long-term anxiety and physiological consequences of regular exposure to media violence in adolescence.
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Media Violence and Children's ADHD-Related Behaviors: A Genetic Susceptibility Perspective
Sanne Nikkelen et al.
Journal of Communication, February 2014, Pages 42–60
Abstract:
This study examined the relationship between media violence exposure and Attention-deficit/hyperactivity disorder (ADHD)-related behaviors. Using survey (parent-reported) and genetic data of 1,612 Dutch children (aged 5 to 9 years), we examined genetic disposition as a possible cause of individual differences in children's use of and susceptibility to media violence. The gene variant of interest was the 5-HTTLPR polymorphism, which has been associated with ADHD-related behaviors in previous research. Results showed that the “long” variant of the gene polymorphism was related to greater violent media use, which in turn was related to more ADHD-related behaviors. The 5-HTTLPR genotype did not moderate the effect of media violence on ADHD-related behaviors. This study provides insight into the role of genetic factors in media effects.
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Extending Role Congruity Theory of Prejudice to Men and Women With Sex-Typed Mental Illnesses
Anne Koenig & Alice Eagly
Basic and Applied Social Psychology, January/February 2014, Pages 70-82
Abstract:
We tested role congruity theory, which states that prejudice arises from an incongruity between group stereotypes and role characteristics, by assessing prejudice toward men and women with a masculine or feminine mental illness. Across two studies, participants acting as a vocational counselor rated the suitability of each target individual in each role. Men and individuals with a masculine sex-typed illness were more suitable for agentic roles, whereas women and individuals with a feminine sex-typed illness were more suitable for communal roles. In addition, sex and mental illness sex-type were better predictors of prejudice than evaluations of the group.
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Rotem Saar-Ashkenazy et al.
Journal of Traumatic Stress, February 2014, Pages 18–26
Abstract:
Memory deficits are a common complaint of patients with posttraumatic stress disorder (PTSD). Despite vivid trauma-related memory, previous studies report memory impairment for nontrauma-related stimuli when compared to controls, specifically in associative memory (Guez et al., 2011). Healthy individuals show hemispheric memory asymmetry with left-prefrontal lateralization of encoding and right-prefrontal lateralization of episodic retrieval, suggesting a role for interhemispheric communication in memory-related tasks (Gazzaniga; Ringo, Doty, Demeter, & Simard). Because brain magnetic resonance imaging (bMRI) studies in PTSD patients report volume changes in various regions, including white matter and corpus callosum (CC), we aimed to test the relationship between memory deficits and CC volume in PTSD patients. We probed for specific alterations in associative memory in PTSD and measured the volume of subportions within the CC employing bMRI. Our main finding was a reduction in CC white-matter volume in PTSD patients, as compared to controls, t(35) = −2.7, p = .010, that was correlated with lower associative performance (r = .76, p = .003). We propose that CC volume reduction is a substrate for the associative memory deficits found in PTSD.
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Sarah Thompson et al.
Psychoneuroendocrinology, forthcoming
Abstract:
Maternal depression serves as a potent source of stress among offspring, greatly enhancing the risk of numerous adverse outcomes including youth depression. Several factors moderate the transmission of depression from mothers to offspring. However, the role of genetic characteristics in this process merits further exploration. Consistent with an interpersonal perspective on depression, the present study focused on a genetic polymorphism that has been shown to be relevant to social functioning, the rs53576 polymorphism of the oxytocin receptor gene (OXTR). In a community sample of 441 youth, OXTR genotype moderated the association between maternal depression in early childhood and youth depressive symptoms in adolescence, such that youth possessing at least one A allele of OXTR who also had a history of maternal depression exhibited the highest levels of depressive symptoms at age 15. In order to explore possible interpersonal mediators of this effect, conditional process analyses examined the role of youth social functioning in adolescence. Results suggest that OXTR genotype may partially account for the transmission of maternal depression to youth and support the role of dysfunctional social processes as a mechanism through which OXTR influences the development of depressive symptoms.
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Early life stress modulates oxytocin effects on limbic system during acute psychosocial stress
Simone Grimm et al.
Social Cognitive and Affective Neuroscience, forthcoming
Abstract:
Early life stress (ELS) is associated with altered stress responsivity, structural and functional brain changes and an increased risk for the development of psychopathological conditions in later life. Due to its behavioral and physiological effects, the neuropeptide oxytocin (OXT) is a useful tool to investigate stress responsivity, even though the neurobiological underpinnings of its effects are still unknown. Here we investigate the effects of OXT on cortisol stress response and neural activity during psychosocial stress. Using functional magnetic resonance imaging in healthy subjects with and without a history of ELS, we found attenuated hormonal reactivity and significantly reduced limbic deactivation after OXT administration in subjects without a history of ELS. Subjects who experienced ELS showed both blunted stress reactivity and limbic deactivation during stress. Furthermore, in these subjects OXT had opposite effects with increased hormonal reactivity and increased limbic deactivation. Our results might implicate that reduced limbic deactivation and HPA-axis responsivity during psychosocial stress are markers for biological resilience after ELS. Effects of OXT in subjects with a history of maltreatment could therefore be considered detrimental and suggest careful consideration of OXT administration in such individuals.